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作者最近遇到一例室上性心动过速患者,推测其折返机理可能有结室型Mahaim氏纤维和隐匿性房室旁道参与,特报道于下。临床和心电图资料患者女性,19岁,因近6年来反复发作心悸、胸闷而于1984年9月10日入院。劳累、精神紧张、睡眠不佳、月经来潮或感冒均为心动过速发作的诱因;每次发作历时数秒至十几小时不等;偶尔,平卧或深吸气可使发作中止。于6年前发作时心电圈检查诊断为室上性心动过速。曾应用过利多卡困,西地兰、心得安、心得宁、安定、慢心律和异搏停等药物,均未能控制心动过速的发作,发病以来无晕厥或抽搐发生。体检除有心律不齐外,无任何异常发现。入院后经口服双异丙吡胺(0.2,每日四次)和阿托品(0.3mg,每日三次),发作得到控制,保持频率在60—70次/分的窦性心律。于1984年9月20日出院。
The authors recently encountered a case of supraventricular tachycardia, presumably the mechanism of its reentry may have conjunctival Mahaim fibers and occult atrioventricular access, especially reported in the next. Clinical and electrocardiographic data The patient, 19 years old, was admitted to hospital on September 10, 1984 due to recurrent palpitations and chest distress in the past 6 years. Tired, nervous, poor sleep, menstrual cramps or cold are the triggers of tachycardia; each attack lasted from a few seconds to more than ten hours; occasionally, supine or deep breathing can make the seizure stop. Echocardiography was diagnosed as supraventricular tachycardia 6 years ago. Had used Lidoca stuck, cedilanid, propranolol, experience Ning, stability, slow heartbeat and verapamil and other drugs, failed to control the onset of tachycardia, onset no syncope or convulsions. Apart from arrhythmia physical examination, without any abnormal findings. After admission, oral dipicidin (0.2, four times a day) and atropine (0.3 mg, three times a day) were administered and the seizures were controlled and sinus rhythm was maintained at a frequency of 60-70 beats / min. Discharged on September 20, 1984.