瑞士实验肿瘤研究所的研究人员最近报道 ,人的腺联病毒 AAV(adeno- associated virus)能选择性诱导那些缺少活性 P5 3蛋白的肿瘤细胞凋亡。当前对肿瘤的治疗大多数是通过放射线或化学药物破坏细胞的 DNA。在有 DNA损伤情况下 ,正常细胞内有活性的 P5 3肿瘤抑制蛋白可通过使细胞 Researchers at the Swiss Institute of Experimental Oncology recently reported that human adeno-associated virus AAV (AAV) selectively induces apoptosis in tumor cells that lack active P53 protein. The current treatment of tumors is mostly the destruction of cells’ DNA by radiation or chemical drugs. In the presence of DNA damage, the active P5 3 tumor suppressor protein in normal cells can be