【摘 要】
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Acute coronary syndrome (ACS) is associated with sudden,reduced blood flow to the heart and usually correlated with coronary thrombosis [1].Therefore,platelets
【机 构】
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Department of Urology, Affiliated Hospital of Nantong University, Nantong 226000, China“,”Department
论文部分内容阅读
Acute coronary syndrome (ACS) is associated with sudden,reduced blood flow to the heart and usually correlated with coronary thrombosis [1].Therefore,platelets play an important part in the pathogenesis and complications of ACS.Platelet activation is a complex event involving activation of intracellular signaling cascades,such as calcium signaling,which leads to platelet activation,secretion and aggregation.Platelets are highly specialized secretory cells that have a well characterized patte of cell activation and intracellular signaling.Platelet activation causes increased cytosolic Ca2+ concentration,which could be limited by accelerating sarcoplasmic/endoplasmic reticulum calcium ATPase (SERCA)activity [2].SERCA activity can be regulated by Ca2+/calmodulin-dependent protein kinase Ⅱ (CaMKII) [3].Furthermore,it has been shown that CaMKII is increased in activated platelets,indicating that cytosolic Ca2+ concentration is crucial to induce platelet activation [4].
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