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目的探讨一氧化碳中毒后迟发性脑病的临床特点与防治,以期提高临床医生对本病的认识和防治效果。方法对我院2004年至2007年收治的一氧化碳中毒后迟发性脑病的临床资料进行回顾性研究。结果几乎所有患者脑部均散在点状或片状病灶,多分布在脑白质和苍白球处,大部分患者有脑电图中度或重度异常,临床主要表现为进行性智能减退、失语、球麻痹、大小便失禁或精神错乱等症状。结论多数迟发性脑病患者发生在一氧化碳中毒后2-30天,治疗以高压氧、抗脑水肿、活化脑细胞为主,预后较差,重在预防。
Objective To explore the clinical features and prevention and treatment of delayed encephalopathy after carbon monoxide poisoning in order to improve clinicians awareness of the disease and prevention and treatment. Methods The clinical data of delayed encephalopathy after carbon monoxide poisoning admitted to our hospital from 2004 to 2007 were retrospectively studied. Results Almost all patients were scattered in the brain spot or flaky lesions, mostly in white matter and globus pallidus, most patients with moderate or severe EEG abnormalities, the main clinical manifestations of progressive intellectual decline, aphasia, the ball Paralysis, incontinence or insanity and other symptoms. Conclusions Most patients with delayed encephalopathy occur 2-30 days after carbon monoxide poisoning. The treatment is mainly hyperbaric oxygen, anti-brain edema, and brain cell activation. The prognosis is poor and the emphasis is on prevention.