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肾小球肾炎是机体免疫调节功能异常，形成大量ＩＣ，在机体排除ＩＣ功能低下时，ＩＣ沉积于肾脏而致病。本实验结果证明，肾小球肾炎病人血清中ＣＩＣ、ｓＩＬ－２Ｒ显著高于正常人，Ｐ＜０．０１，ＣＭＣＳ显著低于正常人，Ｐ＜０．０１，ＴＮＦ、ＩＦＮ显著高于正常人，Ｐ＜０．０１，ＩＬ－８与ＩＬ－２两者无显著差异，Ｐ＜０．０５。从而证明，肾炎病人清除ＩＣ功能不强，血清中高水平的ＣＩＣ沉积于肾脏，引起肾脏损伤。
Glomerulonephritis is an abnormal immune function of the body, forming a large number of IC, IC function in the body to exclude low IC deposition in the kidney and pathogenic. The results of this experiment demonstrated that the serum levels of CIC and sIL-2R in patients with glomerulonephritis were significantly higher than those in normal controls (P <0.01), and CMCS was significantly lower than that in normal controls (P <0.01, TNF and IFN) , P <0.01, there was no significant difference between IL-8 and IL-2, P <0.05. Proved that nephritis patients with IC function is not clear, high serum levels of CIC deposited in the kidneys, causing kidney damage.