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ＥＡＣ或Ｓ１８０鼠分别ｉｇ４０、８０和１２０ｍｇ／ｋｇ·ｄ－１的ＴＰ对ＥＡＣ的抑制率分别为６５．３％、５１．０％和４８．７％；对Ｓ１８０的抑制率为６６．３％、６８．５％和５９．８％。ｉｇＴＰ的ＥＡＣ鼠的胸腺指数比对照组分别提高５０．７％、４０．６％和１９．８％；脾指数提高３４．４％、３１．１％和２０．０％；Ｓ１８０鼠的胸腺指数也分别提高了６６．２％、４７．２％和４８．１％；脾指数略有增高。离体细胞培养测定ＴＰ对致敏Ｔ淋巴细胞的粘瘤和钻瘤活性结果表明：各ＴＰ组ＥＡＣ鼠的胸腺或脾淋巴细胞对自体瘤细胞的ＡＴＩ和ＥＴＩ均明显高于对照组（Ｐ＜０．０１）。正常小鼠ｉｇ ６０ｍｇ／ｋｇ·ｄ－１的ＴＰ，连续给药１２ｄ后、腹腔Ｍ吞噬ＣＲＢＣ的百分率和吞噬指数比对照组分别增加６９．６％和６８．７％。
EAC or S180 mice had ig40, 80 and 120mg/kg·d-1 TP inhibition rate of EAC were 65.3%, 51.0% and 48.7%, respectively; inhibition rate of S180 was 66.3% , 68.5% and 59.8%. The thymus index of igTP EAC mice was increased by 50.7%, 40.6%, and 19.8%, respectively; the spleen index was increased by 34.4%, 31.1%, and 20.0%; the thymus index of S180 mice. They also increased by 66.2%, 47.2%, and 48.1%, respectively; the spleen index increased slightly. The results of in vitro cell culture assay of TP on the sensitized T lymphocyte myxomatous and tumor-bearing activity showed that the thymic or splenic lymphocytes of TP groups were significantly higher in thymic or spleen lymphocytes than in the control group (P< 0.01). The percentage of TP of normal mouse ig 60 mg/kg·d-1 after continuous administration for 12 days and peritoneal M engulfment of CRBC increased by 69.6% and 68.7%, respectively, compared with the control group.