单侧声带麻痹(VCP)可导致不同程度的声嘶和吞咽困难,为消除症状,并纠正麻痹声带内收功能不良所致声门裂隙。与目前广泛应用的声带注射或甲状软骨成形术等静态治疗相比,麻痹声带的再活动术更符合生理。由于神经-神经吻合术或神经肌蒂移植术等动态治疗尚未能广泛用于临床,本文介绍一种通过环甲肌(CT)的电起搏刺激麻痹声带再活动的新技术。作者检查了萎缩肌肉对电刺激的反应性以及狗在失神经支配15个月后对电起搏反应中肌收缩的效能。实验:①分别用压榨、重复缝扎和切断3cm神经的方法损伤喉返神经。在 Unilateral vocal cord paralysis (VCP) can lead to varying degrees of hoarseness and difficulty swallowing in order to eliminate the symptoms and correct vocal fold fissures due to poor adduction of the paralyzed vocal cord. With the current widespread application of vocal cord injection or thyroid cartilage and other static treatment, paralysis vocal cord reactivation more in line with physiology. Because dynamic therapy such as nerve-nerve anastomosis or neuromuscular pedicle transplantation has not been widely used in clinical practice, this paper introduces a new technique of reactivation of paralyzed vocal cord by electrical pacing of cricothyroid muscle (CT). The authors examined the responsiveness of shrunken muscles to electrical stimulation and the efficacy of dogs in contracting electrical pacing reactions after 15 months of denervation. Experiments: 1, respectively, by pressing, repeated suture and cut off 3cm nerve damage recurrent laryngeal nerve. in