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马兜铃酸肾病 (aristolochicacidnephropathy ,AAN)发病机制目前尚不十分清楚 ,细胞免疫是否参与疾病的启动过程 ,国内外尚有争论。虽然一般认为 ,AAN为少细胞性的间质纤维化 ,但其病理证据来自于肾移植后的摘除肾[1] 。近年研究发现 ,肾间质中可见CD4 + 、CD8+ 和CD6 8+ 细胞?
The pathogenesis of aristolochic acid nephropathy (AAN) is not yet fully understood. Whether cellular immunity is involved in the initiation of the disease remains controversial at home and abroad. Although it is generally accepted that AAN is a subset of interstitial fibrosis, its pathological evidence is derived from the removal of kidneys after renal transplantation [1]. Recent studies found that renal interstitial CD4 +, CD8 + and CD6 8+ cells?