论文部分内容阅读
急性或陈旧性声损伤除造成听力下降外,多数情况下尚能引起严重的高频耳鸣,患者很是痛苦。Habermann等许多学者研究表明,声损伤可使耳蜗产生机械性损害,基础代谢和氧耗量增加,血管收缩和血栓形成,从而引起局部缺氧,最终导致耳蜗感觉细胞的改变。本文报告作者们1980年3～9月间应用76%泛影葡胺等治疗急性和陈旧性声损伤的结果。共治疗急性声损伤4例,病史4周以上的陈旧性声损伤16例。患者均经临床、纯音和语言测听等听力学检查。20例中除1例6000赫听力损失在20～40dB,其他病例4000、6000和8000赫频段听力损失均大于50dB。方法:10%Rheomacrodex 500ml加入Complamin 0.5～2.0g输注,隔日一次,
In addition to causing acute hearing loss or old sound damage, in most cases still can cause severe high-frequency tinnitus, the patient is very painful. Habermann and many other scholars have shown that acoustic damage can cause mechanical damage to the cochlea, basal metabolism and oxygen consumption increased, vasoconstriction and thrombosis, causing local hypoxia, eventually leading to changes in cochlear sensory cells. This article reports the authors’ treatment of acute and old acoustic lesions with 76% diatrizoate between March and September 1980. A total of 4 cases of acute acoustic injury, history of more than 4 weeks old sound damage in 16 cases. Patients were clinically, pure tone and audiometry and other audiological examination. In 20 cases, the hearing loss of one case of 6000 Hz was 20 ~ 40 dB, while that of the other cases of 4000, 6000 and 8000 Hz was greater than 50 dB. Methods: 10% Rheomacrodex 500ml Complamin 0.5 ~ 2.0g infusion, once every other day,