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应用生物化学方法测定了４０例小儿肺炎血浆血小板活化因子（ＰＡＦ），结果急性期患儿血ＰＡＦ为５．０８±１．０２ｍｇ／Ｌ，明显高于正常对照组（１．４５±０．７９ｍｇ／Ｌ），恢复期时为１．１１±０．３８ｍｇ／Ｌ，已降至正常。本研究还发现伴有喘鸣的肺炎患儿血浆ＰＡＦ值较普通型肺炎患儿高，血白细胞总数与皿浆ＰＡＦ呈正相关。说明ＰＡＰ不仅是一种过敏介质，更是一种炎症介质。作者推测肺内产生较低量ＰＡＦ可导致肺内非特异性炎症，产生较高量时则引发气道高反应性，结果提示抗炎或抗ＰＡＰ是治疗肺部炎症疾病的基础。
40 cases of children with pneumonia were assayed for the platelet-activating factor (PAF) by biochemical methods. Results The blood PAF in the acute stage was 5.08 ± 1.02mg / L, which was significantly higher than that of the normal control group (1.45 ± 0.79mg / L), recovery period of 1.11 ± 0.38mg / L, has dropped to normal. This study also found that children with pneumonia associated with wheezing plasma PAF values higher than children with ordinary pneumonia, the total number of leukocytes and dish PAF was positively correlated. PAP is not only an allergic medium, but also an inflammatory mediator. The authors speculate that a lower amount of PAF in the lungs leads to nonspecific inflammation in the lungs and a higher amount in response to airway hyperresponsiveness, suggesting that anti-inflammatory or anti-PAP is the basis for the treatment of inflammatory diseases of the lungs.