论文部分内容阅读
最近20年在处理心肌梗塞心源性休克方面取得了重大进展,此类病人存活率从15％上升为50％以上。这些进展为许多新技术如主动脉内气囊反搏术,冠状动脉旁路术,冠状动脉溶栓术和经皮冠状动脉成形术等广泛应用的结果。本文着重论述急性心肌梗塞心源性休克的致病机理和处理现状。 一、致病机理 心肌梗塞心源性休克的三种致病机理为:(一)广泛地左室收缩功能障碍。(二)广泛地右室梗塞。(三)心肌梗塞引起的左或右室机械功能障碍。在第一种致病机理中又包括下述四
In the recent two decades, significant progress has been made in the treatment of cardiogenic shock of myocardial infarction. The survival rate of such patients has risen from 15% to over 50%. These advances are the result of a wide range of new technologies such as intra-aortic balloon counterpulsation, coronary artery bypass surgery, coronary thrombolysis and percutaneous transluminal coronary angioplasty. This article focuses on the pathogenesis and treatment of cardiogenic shock in acute myocardial infarction. First, the pathogenic mechanism Three pathogenic mechanisms of myocardial infarction cardiogenic shock: (a) a wide range of left ventricular systolic dysfunction. (B) a wide range of right ventricular infarction. (C) myocardial infarction caused by left or right ventricular mechanical dysfunction. In the first pathogenic mechanism also includes the following four