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选择经颅脑ＣＴ扫描证实为单侧皮层下病变患者６９例，在发病２周，１月及２月分别进行失语症检查。同时，进行ＢＥＡＭ及ＣＴ图象标准化处理。结果显示，皮层下失语患者病灶多在基底节区偏外侧，偏前部及偏上部。病变偏外侧者及丘脑性失语者脑电地形图异常率高，且此类患者多有较重的听理解障碍，而尾状核受损者失语中有明显的构音障碍及音韵障碍。作者讨论了皮层下失语的临床特点，并认为导致失语的机制多与皮层下病变直接或间接影响皮层语言区（血流量减少、代谢低下）有关，但尾状核似可作为言语的皮层下整合中枢，丘脑可作为皮层下语言中枢，在部分患者的失语中起作用。
Sixty-nine patients with unilateral subcortical lesions confirmed by CT scans were selected and examined for aphasia at 2 weeks, 1 month and 2 months respectively. At the same time, BEAM and CT image standardization. The results showed that patients with subcortical aphasia lesions more in the lateral basal ganglia, prefrontal and partial upper. Ectopic lateral hypothalamus and hypothalamus EEG topography abnormal rate, and such patients have more severe hearing disorders, and atelectasis in caudate nucleus significant dysarthria and phonological disorders. The authors discuss the clinical features of subcortical aphasia and consider that the mechanisms that lead to aphasia are often associated with subcortical lesions that directly or indirectly affect the cortical language regions (reduced blood flow, hypometabolism), but caudate nuclei may serve as a cortical sublingual integration The thalamus and thalamus serve as a subcortical language center and play a role in the aphasia of some patients.