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甲状腺纤维化是一个重要的病理生理过程,由于各种原因如持续性感染、自身免疫反应、化学刺激、辐射或机械损伤等刺激下,均可以出现纤维性改变。其特征是以组织的过度增生和肿大,滤泡间纤维性改变为特点,最终可导致结构破坏和功能减退[1]。在甲状腺的病理过程中,纤维化改变以甲状腺炎的某些类型表现相对突出,如桥本甲状腺炎纤维化亚型(fibrous variant of Hashimoto’s thyroiditis,FVHT)、Riedel’s甲状腺炎(Riedel’s thyroiditis,RT)和亚急性肉芽肿
Thyroid fibrosis is an important pathophysiological process, due to various reasons such as persistent infection, autoimmune reaction, chemical irritation, radiation or mechanical damage and other stimuli, can appear fibrous changes. Its characteristics are characterized by hyperplasia and enlargement of the tissue and fibrous changes between the follicles, which eventually lead to structural damage and dysfunction [1]. In the pathological course of the thyroid, changes in fibrosis are relatively prominent in certain types of thyroiditis, such as fibrous variant of Hashimoto’s thyroiditis (FVHT), Riedel’s thyroiditis (RT), and Subacute granuloma