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目的观察急性脑梗死丁苯酞的抗炎作用。方法选择80例急性脑梗死患者随机分为治疗组和对照组各40例,均给予抗血小板聚集、活血化瘀药物,治疗组加用丁苯酞;检测治疗前、治疗后血清超敏C反应蛋白水平(hs-CRP)变化,并对神经功能缺损进行评分。结果治疗后7d、14d治疗组hs-CRP水平低于对照组(P<0.05),7d、14d治疗组神经功能缺损评分低于对照组(P<0.05)。结论丁苯酞可显著降低血清hs-CRP水平,起到抗炎作用。
Objective To observe the anti-inflammatory effect of butylphthalide in acute cerebral infarction. Methods Eighty patients with acute cerebral infarction were randomly divided into treatment group and control group, 40 cases were given anti-platelet aggregation, blood circulation and stasis drugs, the treatment group with butylphthalide; before and after treatment, serum hypersensitivity C reaction Protein levels (hs-CRP) changes, and neurological deficits were scored. Results The levels of hs-CRP in the treatment group were lower than those in the control group on the 7th day and the 14th day (P <0.05). The neurological deficits in the treatment group on the 7th and 14th day were lower than those of the control group (P <0.05). Conclusion Butylphthalide can significantly reduce serum hs-CRP levels, play an anti-inflammatory effect.