妊娠高血压可伴有循环血小板数量减少,血小板机能障碍可能参与高血压发生。有人证明高危患者应用抗血小板治疗有益。一25岁孕妇,36周时发生高血压,同意在妊娠期进行体外血小板聚集的纵向研究。患者系第三次妊娠。第一次足月顺产一健康婴儿,妊娠期顺利;第二次孕9周时自然流产。无吸烟及相关既往史,亦无预痫或高血压家族史。于孕22、28和35周时行超声检查正常。孕35周时血压轻度升高,但血小板计数明显减少(孕28周223×10~(-9)/L至孕35周为135×10~(-9)/L)。在医院,血压最高为150/110mmHg,为控制血压,间断静 Pregnancy-induced hypertension can be associated with reduced circulating platelets, and platelet dysfunction may be involved in the development of hypertension. It has been shown that antiplatelet therapy is beneficial in high-risk patients. A 25-year-old pregnant woman has hypertension at 36 weeks and agrees with a longitudinal study of in vitro platelet aggregation during pregnancy. The patient is the third pregnancy. The first full-term birth of a healthy baby, pregnancy smooth; the second 9 weeks of spontaneous abortion. No history of smoking and related history, no family history of pre-eclampsia or hypertension. At 22, 28 and 35 weeks of pregnancy, ultrasound examination was normal. Blood pressure was slightly elevated at 35 weeks of gestation, but the platelet count was significantly reduced (223 × 10 ~ (-9) / L at 28 weeks to 135 × 10 ~ (-9) / L at 35 weeks gestation). In the hospital, blood pressure is up to 150 / 110mmHg, for the control of blood pressure, intermittent static