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焦化工人肺癌死亡超出一般居民11.29倍。其肺癌危险主要来自煤的不完全燃烧产物—以苯并[a]芘为代表的致癌性多环芳烃(PAH)。当其进入机体后,经代谢,分别由肝、胆和肾脏排泄。用尿浓缩物做致突变试验,可发现人体对致癌性多环芳烃的接触水平。 研究对象与方法 研究对象为太钢焦化厂满1年工龄的工人,共55名。其中重污染作业38名(吸烟30名,不吸烟8名),轻污染作业17名(吸烟13名,不吸烟4名)。重污染作业选焦炉和成品车间,轻污染作业选机关和机电车间。对照选自某省直机关35岁以上男性职工,共7名。
Coking workers lung cancer deaths 11.29 times more than the average resident. The risk of lung cancer comes mainly from the incomplete combustion products of coal - the carcinogenic polycyclic aromatic hydrocarbons (PAHs) represented by benzo [a] pyrene. When it enters the body, the metabolism, respectively, by the liver, gall bladder and kidney excretion. Mutagenicity test with urine concentrates can be found in human exposure to carcinogenic PAHs. Research object and method The research object is a total of 55 workers in TISCO Coking Plant who have completed one year of service. Among them, 38 were heavily polluting (30 smokers, 8 non-smokers) and 17 were light polluting workers (13 smokers and 4 non-smokers). Heavy polluting operation of the coke oven and the finished product workshop, light pollution operation of the selected agencies and mechanical and electrical workshop. Control selected from the provincial organs over the age of 35 male workers, a total of seven.