论文部分内容阅读
病历报告:男患,54岁。住院号7155。三十年前曾患肺结核,进行抗痨治疗后病情稳定。去年8月初,病人自感咳嗽、胸闷、气急加剧,稍活动后更加明显,经当地医院检查,胸透为右侧气胸,行抗痨治疗,经胸腔穿刺抽气多次,未见好转,于1983年9月20日转来本院。体检:T36.8℃,P 80次/分,R20次/分,Bp168/94mmHg。慢性病容,稍气急,气管左移。右胸:肋闻隙增宽,呼吸运动减弱,语颤减弱,叩诊鼓音,语音传导减弱;左胸:呼吸音增强。胸片(2698号)显示右上肺结核大部硬结,右侧气胸,压缩肺组织约80%。在常规消毒和普鲁卡因局麻下,用胸腔穿刺针
Medical records: Male suffering, 54 years old. Hospital number 7155. Thirty years ago had tuberculosis, anti-tuberculosis treatment after a stable condition. Early August last year, the patient felt cough, chest tightness, aggravated urgency, after a little more obvious activity, the local hospital examination, chest through the right pneumothorax, anti-tuberculosis treatment, aspiration through the chest puncture many times, did not improve, at September 20, 1983 transferred to the hospital. Physical examination: T36.8 ℃, P 80 beats / min, R20 beats / min, Bp168 / 94mmHg. Chronic disease, a little shortness of breath, tracheal left. The right chest: rib gap widened, decreased respiratory activity, weakened tremor, percussion drum sound, voice conduction weakened; left chest: increased respiratory sounds. The chest radiograph (No. 2698) showed most of the right upper pulmonary tuberculosis induration, the right pneumothorax, compressed lung tissue by about 80%. In the conventional disinfection and procaine local anesthesia, with thoracentesis needle