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目的 :探讨造成髋关节发育不良的病理发病机制。方法 :将 18只幼兔的左髋关节屈曲膝关节伸直位石膏固定 4周 ,比较双侧髋关节的X线片及病理组织学变化。结果 :髋关节屈曲膝关节伸直位石膏固定 4周造成了髋关节发育不良的动物模型。髋臼变浅、股骨头变小、向外上方移位。X线片可见髋臼指数增大 ,由固定前的 (2 8.44± 3 .91)°增大为 (3 3 .17± 3 .65 )° ;臼头指数减小 ,由固定前的 (75 .2 0± 4.41) %减小为 (5 8.71± 8.2 1) %。组织学观察可见关节软骨退行性改变。结论 :髋关节屈曲膝关节持续伸直位状态 ,容易造成髋关节发育不良。随着时间的延长 ,病理变化加重。及早复位 ,髋关节可以恢复正常
Objective: To investigate the pathogenesis of dysplasia of the hip. Methods: 18 young rabbits’ left hip joint flexion and extension orthostatic gypsum were fixed for 4 weeks, the bilateral hip joint radiography and histopathological changes were compared. RESULTS: Hip Buckling Knee extension of the plaster for 4 weeks resulted in an animal model of hip dysplasia. Acetabular shallow, smaller femoral head, outward shift above. The X-ray showed that the acetabular index increased from (2 8.44 ± 3.91) ° to (3 3 .17 ± 3.65) ° before fixation and decreased from (75 .2 0 ± 4.41)% to (5 8.71 ± 8.2 1)%. Histological observation showed that articular cartilage degenerative changes. Conclusion: The flexion and extension of the knee joint continues to be straight, easily lead to dysplasia of the hip joint. With the extension of time, pathological changes aggravate. Early reset, the hip can return to normal