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本文使用条件—检验刺激技术,研究了电刺激杏仁皮质内侧核对猫丘脑后核内脏大神经诱发放电的影响。电刺激杏仁皮质内侧核抑制丘脑后核的中等和长潜伏期内脏大神经诱发放电呈现两种形式:即时抑制和迟发抑制。前者可能是通过从杏仁核到丘脑后核的神经通路产生的,静脉注射纳洛酮可消除这种抑制,提示有鸦片样物质的参与。后者可能涉及神经—体液性因素。结果提示,杏仁核参与抑制内脏痛。
In this paper, the condition-test stimulus was used to study the effect of electrical stimulation of the medial amygdaloid nucleus on the visceral nociceptive firing in the posterior nucleus of the thalamic nucleus. Electrical stimulation of the medial amygdaloid nucleus to inhibit the posterior thalamic nucleus of the medium and long latent period induced visceral nerve-induced discharge showed two forms: immediate and delayed inhibition. The former may be produced through the neural pathway from the amygdala to the posterior nucleus of the nucleus. Intravenous naloxone abolished this inhibition, suggesting the involvement of opioids. The latter may involve neuro-humoral factors. The results suggest that amygdala participates in the inhibition of visceral pain.