治疗类风湿性关节炎(RA)的药物目前主要有两类。一类是非甾体类抗炎免疫药(如阿斯匹林、消炎痛、布洛芬等)。另一类是甾体类抗炎免疫药(即肾上腺皮质激素类).这两类药物均可抑制PGs合成。由于PGs在RA发病中不仅有致炎的一面,也有抗炎的一面,且PGs又是骨质蛋白合成的重要辅助因子,这些药物抑制PGs合成,虽可使RA炎症得到控制,但并不能阻止骨质病变的发展。因此,抗炎治疗对于RA利少 There are currently two main types of drugs for the treatment of rheumatoid arthritis (RA). A class of non-steroidal anti-inflammatory immune drugs (such as aspirin, indomethacin, ibuprofen, etc.). The other is steroidal anti-inflammatory immune drugs (ie, adrenocorticotropic hormone), both of which inhibit the synthesis of PGs. Since PGs not only cause inflammation in the pathogenesis of RA, but also have anti-inflammatory side, and PGs is an important cofactor of bone protein synthesis, these drugs inhibit the synthesis of PGs, although can make RA inflammation can be controlled, but does not stop the bone Quality of disease development. Therefore, anti-inflammatory treatment is less for RA