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本文观察窒息新生儿心肌酶谱的动态变化，用以评估窒息对新生儿心肌损害的程度。设对照组15例．窒息组30例，包括宫内窘迫10例（心率＞160次／分），轻度窒息16例，重度室息4例，作心肌酶时间为生后24小时内（30例），3～5天（15例）．＞7天（30例）。结果24小时、3～5天GOT、LDH、CPK、CPK－MB值均明显高于对照组（P＜0．01）。窒息组治疗前后对比呈逐渐下降趋势。CPK的同功酶（CPK－MB）本组窒息组（47.83±21．9）明显高于对照组（12．06±6．42），在心肌损害判断上更有重要意义。有助于早期预测心功能的损害和及对治疗，以免进一步发展为心衰、心源性休克和心肌坏死等不可逆现象。
This article observed changes in myocardial enzymes of asphyxia neonates to assess the extent of neonatal myocardial damage caused by asphyxia. The control group of 15 cases. 30 cases of asphyxia group, including 10 cases of intrauterine distress (heart rate> 160 beats / min), mild asphyxia in 16 cases, severe ventricular infarction in 4 cases, myocardial enzyme time within 24 hours after birth (30 cases), 3 to 5 Day (15 cases). > 7 days (30 cases). Results The values of GOT, LDH, CPK and CPK-MB in 24 hours and 3 to 5 days were significantly higher than those in control group (P <0.01). Asphyxia group before and after treatment showed a gradual downward trend. CPK isoenzyme (CPK-MB) asphyxia group (47.83 ± 21.9) was significantly higher than the control group (12.06 ± 6.42), in the judgment of myocardial damage is more important. Help to predict the damage of early cardiac function and treatment, so as to avoid the further development of irreversible phenomena such as heart failure, cardiogenic shock and myocardial necrosis.