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目的为了探讨新生儿窒息所致中枢神经元缺氧性损伤及耳蜗毛细胞功能的损害对新生儿早期听力的影响;和评价DPOAE、NBNA在其中价值。方法对36例新生儿窒息进行畸变产物耳声发射及NBNA评价。结果正常新生儿的耳声发射与NBNA评分中行为能力3项通过时间比较无显著性差异P>0.05;在正常与窒息新生儿及轻度与重度窒息中,比较NBNA中行为能力3项与耳声发射通过时间均有显著性差异P<0.001。结论提示正常新生儿耳声发射与NBNA评分中的行为能力3项通过基本同步;新生儿窒息所导致耳蜗毛细胞功能及中枢神经元的损害恢复不同步;窒息越严重,耳蜗毛细胞功能及听觉中枢神经元功能恢复越慢。
Objective To investigate the neonatal asphyxia-induced central neuronal hypoxia damage and cochlear hair cell dysfunction in early neonatal hearing impact; and evaluation of DPOAE, NBNA in which the value. Methods Distortion otoacoustic emissions and NBNA were evaluated in 36 neonates with asphyxia. Results There was no significant difference in the viability between the normal newborn with otoacoustic emissions and the NBNA score (P> 0.05). Among the normal and asphyxiated neonates and mild and severe asphyxia, Acoustic emission transit time was significantly different P <0.001. The results suggest that normal neonatal otoacoustic emissions and NBNA score in the three basic synchronized viability; neonatal asphyxia caused by cochlear hair cell function and central neuron damage recovery asynchronous; more serious asphyxia, cochlear hair cell function and hearing Slower central nervous system function recovery.