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目的探讨影响青少年焦虑敏感的内在机制,为早期心理和行为干预提供理论支持。方法设研究组和对照组。研究组共42例为焦虑敏感青少年,对照组30例为焦虑组。分别对其进行症状自评量表(SCL-90)及汉密尔顿焦虑量表(HAMA),脑功能检查评定对比分析。结果焦虑敏感青少年的a波竞争高主频个数(11Hz以上)及主频率(11Hz、12Hz)显著高于对照组(t=5.2,6.17,5.3;P<0.01);熵值和主次频波动度及总谱系差异无显著性,但少氧状况和前后功率逆转数比较显著低于对照组(t=2.41,2.4;P<0.05)。两组的SCL-90及HAMA总分和各因子分除躯体化(t=2.34,P<0.05)有差异外,其余差异无显著性。结论焦虑敏感青少年脑电α主频的高频性可能是青少年焦虑敏感的生物学基础之一,对事件刺激表现为敏感和易唤醒泛化机制。泛化来自于灾难性认知扭曲。
Objective To explore the intrinsic mechanism of affecting anxiety in adolescents and to provide theoretical support for early psychological and behavioral interventions. Methods Study group and control group. In the study group, 42 cases were anxiety-sensitive adolescents, and 30 cases in the control group were anxiety group. Respectively, the Symptom Checklist 90 (SCL-90) and the Hamilton Anxiety Scale (HAMA), brain function test evaluation comparative analysis. Results The frequency of a-wave high frequencies (11Hz and above) and the dominant frequency (11Hz and 12Hz) of anxiety-sensitive adolescents were significantly higher than those of the control group (t = 5.2,6.17,5.3, P <0.01) There was no significant difference between the two groups in the degree of fluctuation and the total pedigree, but the hypoxia status and power reversal number were significantly lower than those in the control group (t = 2.41,2.4; P <0.05). There was no significant difference between the two groups in the total score of SCL-90 and HAMA and in all factors except somatization (t = 2.34, P <0.05). Conclusion Anxiety-sensitive adolescents with high frequency of EEG alpha frequency may be one of the biological basis of adolescent anxiety-sensitive and susceptible to event stimuli and wake-up generalization mechanism. Generalization comes from catastrophic cognitive distortions.