论文部分内容阅读
自 1991年美国胸科医师学会 /危重病医学会 (ACCP/SCCM)提出全身炎症反应综合征 (SIRS)及多器官功能障碍综合征 (MODS) [1] 的概念后 ,人们对SIRS的发病机制已有较深入研究。本文旨在通过SIRS患儿外周血肿瘤坏死因子 α(TNF α)、白细胞介素 1β(?
Since the concept of systemic inflammatory response syndrome (SIRS) and multiple organ dysfunction syndrome (MODS) [1] proposed by the American College of Chest Physicians / Critical Care Medicine (ACCP / SCCM) in 1991, the pathogenesis of SIRS Have been more in-depth study. The aim of this paper is to investigate the relationship between tumor necrosis factor α (TNFα), interleukin-1β