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心肌损害全貌变幻极大,从功能紊乱到细胞死亡,随冠脉管腔狭窄程度和侧供支应情况而异。功能紊乱(机械电变化)可从传导减漫到电静止,从舒张期强直到收缩期无力.冠脉完全阻塞可迅速地,也可缓慢地发生。如果侧支循环无效,血流被完全阻断,从而引起心肌组织大量坏死,即透壁性心肌梗塞(MI)。不称定性粥样化斑粥样化斑崩解可产生不稳定型心绞痛、MI或心脏猝死。近来提出,粥样化斑的纤维冠裂隙使血液渗入内膜,直接与胆固醇结晶、脂肪
The overall picture of myocardial damage changes dramatically, from dysfunction to cell death, with the extent of coronary stenosis and lateral supply should vary. Dysfunction (mechanical and electrical changes) can diffuse from electrical conduction to electrical rest, from diastolic to systolic weakness. Complete coronary occlusion can occur rapidly and slowly. If collateral circulation fails, blood flow is completely blocked, causing massive necrosis of the myocardium, transmural myocardial infarction (MI). Not known qualitative porridge-like porphyria plaque disintegration can produce unstable angina, MI or sudden cardiac death. It has recently been proposed that the crevicular crevices of the atypical plaques allow the blood to penetrate into the intima, crystallizing directly with cholesterol and the fat