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用小鼠造成四种病理模型:甲状腺素型(体内甲状腺素过多):他巴唑型(甲状腺功能抑制):氢考Ⅰ型(肾上腺皮质激素过多);氢考Ⅱ型(肾上腺皮质机能抑制)。以β激动剂造成血浆 cAMP 升高,以 M 胆碱激动剂造成血浆 cGMP 升高,并以助阳药(附子、肉桂),滋阴药(生地、龟板)作实验性治疗。结果表明,甲状腺素型及氢考Ⅰ型 cAMP 系统反应性升高,滋阴药使之改善,他巴唑型及氢考Ⅱ型 cGMP 系统反应性升高或 cAMP 反应性降低,助阳药使之改善。故初步认为前两种属“阴虚”模型,后两种属“阳虚”模型。
Four pathological models were produced in mice: Thyroxine (too much thyroxine in the body): Tapahazole (inhibition of thyroid function): Hydrogen test type I (adrenocorticotropic hormone); Hydrogen test II (adrenocortical function) inhibition). Plasma β-amyloid (CAMP) levels were increased by β-agonists, plasma cGMP levels were increased by M-cholinergic agonists, and adjuvant therapy (Fuzi, Cinnamon) and Ziyin (reproductive land, turtle shell) were used for experimental treatment. The results showed that the reactivity of thyroxine type and hydrogen proteolytic type I cAMP system was increased, and the noxious yin drugs improved it, and the reactivity of methimazole type and hydrogen type II cGMP system was increased or the cAMP reactivity was reduced, and the positive drug was used. Improvements. Therefore, the first two kinds of genus “Yin virtual” model are preliminarily considered, and the latter two kinds of “yang deficiency” models.