报导Ⅰ的实验材料证明了豚鼠在注射CoCl_2后,虽然有50％的动物产生脂肪肝但与胰岛A细胞或胰腺泡组织所遭受的损害无关,可能是CoCl_2直接影响肝细胞代谢所致。因此过去学者认为胰臓具有抗脂肪肝因素的说法是值得重新考虑的。为了更进一步探讨这个问题,我们又选择了另外一种动物(家兔)来进行实验。1953年Volk The experimental data reported in I report that although CoCl 2 was injected into the guinea pigs, 50% of the animals had fatty liver, which was not related to the damage suffered by pancreatic islet A cells or pancreatic ductal tissue. CoCl 2 may be the direct result of liver cell metabolism. Therefore, in the past, scholars believe that pancreas has anti-fatty liver is a statement worthy of reconsideration. In order to further explore this issue, we have chosen another animal (rabbit) to experiment. 1953 Volk