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目的：探讨心功能改变在高原肺水肿患者发病中的作用。方法：采用六导生理记录仪、切面超声心动图仪及放射免疫法检测了高原肺水肿患者心脏结构、功能、血气及血清内源性洋地黄因子含量，并同高原健康人进行了对比。结果：高原肺水肿患者发病时，外周血中内源性洋地黄因子含量明显下降；肺水肿患者左室舒张末经明显缩小，右室舒张末经、右室流出道及肺动脉内径均明显增宽，射血分数、心搏量、心输出量下降。结论：高原肺水肿患者发病时，存在不同程度的心功能不全。
Objective: To investigate the role of cardiac function changes in the pathogenesis of patients with high altitude pulmonary edema. Methods: The cardiac structure, function, blood gas and endogenous digitalis content in serum of patients with high altitude pulmonary edema were detected by six-lead physiology recorder, echocardiography and radioimmunoassay, and compared with healthy subjects in the plateau. Results: In the patients with high altitude pulmonary edema, the content of endogenous digitalis in the peripheral blood decreased significantly. The left ventricular end-diastolic volume of the patients with pulmonary edema was significantly reduced, and the right ventricular end-diastolic, right ventricular outflow tract and pulmonary artery diameter were significantly increased , Ejection fraction, stroke volume, cardiac output decreased. Conclusion: There are different degrees of cardiac dysfunction in patients with high altitude pulmonary edema.