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氧自由基反应和脂质过氧化反应在正常心肌组织代谢过程中起着重要的作用,且两者处于动态平衡状态。有研究表明,在心肌缺血再灌注损伤过程中,系因产生的大量氧自由基而引发了链式脂质过氧化反应[1]。2007年7月我们研究了硒对家兔心肌缺血再灌注损伤所致的血浆脂质过氧化物丙二醛
Oxygen free radical reactions and lipid peroxidation play an important role in normal myocardial tissue metabolism, and both are in a state of dynamic equilibrium. Studies have shown that in the process of myocardial ischemia-reperfusion injury, due to a large number of oxygen free radicals and trigger chain lipid peroxidation [1]. July 2007 We studied the effects of selenium on myocardial ischemia-reperfusion injury caused by plasma lipid peroxides malondialdehyde