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氨在肝性脑病发病机理上具有关键作用。肝硬化病人的高氨血症被认为由肠菌的尿素酶所产生。幽门螺杆菌(Hp)由于其尿素酶活性较尿素酶阳性肠菌强好多倍而产生大量氨。本研究旨在了解根除Hp对肠菌群得以适当治疗病人的高氨血症的影响。
Ammonia in the pathogenesis of hepatic encephalopathy has a key role. Hypernatremia in cirrhotic patients is thought to be caused by enterococcal urease. Helicobacter pylori (Hp) produces large quantities of ammonia due to its many times more active urease activity than urease-positive enteric bacteria. The aim of this study was to understand the effect of eradication of Hp on hyperammonemia in patients with proper gut microbiota.