在某些类型的外伤之后,角膜易有血管侵入,其中炎性损害更为突出,此时血管反应伴有炎性细胞侵入。有些作者认为某些细胞及其产物有可能刺激新生血管的形成,其中多形核白细胞的侵入可能是主要因素,至少可能起重要作用。前列腺素是炎症反应的有力介质。一些非皮质类固醇的前列腺素抑制剂,如消炎痛,可能以某种方式改变角膜损伤的炎症反应,即一方面可避免皮质类固醇制剂的某些并发症的发生,同时又能有效地抑制角膜的新生血管形成。本报告表明消炎痛对烧伤所致的角膜血管反应有中等度作用。作者使用16只白色家兔进行单盲随机抽样实验,每眼都使用同样的电流烧灼位于角膜上方距 After some types of trauma, the cornea is prone to vascular invasion, in which inflammatory damage is more prominent, when the vascular response accompanied by inflammatory cell invasion. Some authors believe that certain cells and their products may stimulate the formation of new blood vessels, of which infiltration of polymorphonuclear leukocytes may be a major factor, at least possibly playing an important role. Prostaglandins are potent mediators of inflammation. Some non-corticosteroid prostaglandin inhibitors, such as indomethacin, may alter the inflammatory response to corneal injury in a way that avoids some of the complications of corticosteroid preparations on the one hand while effectively inhibiting the corneal Neovascularization. This report shows that indomethacin has a moderate effect on corneal vascular responses to burns. The authors used 16 white rabbits to perform a single-blind randomized sampling experiment using the same current cauterization at each eye