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众多临床和实验研究均证实 ,阻断肾素 血管紧张素系统能明显延缓糖尿病肾病的进展。以前认为 ,血管紧张素Ⅱ(AngⅡ )的生物学作用是通过血管紧张素Ⅱ 1型受体 (AT1)起作用的 ,近年来 ,大量证据表明 ,血管紧张素Ⅱ 2型受体(AT2 )拮抗了几乎大多数AT1受体的生物学作用。为此 ,我
Many clinical and experimental studies have confirmed that blocking the renin-angiotensin system can significantly delay the progress of diabetic nephropathy. It was previously thought that the biological effect of Ang II acts through the AT1 receptor, and in recent years there is ample evidence that AT2 antagonism The biological role of almost all AT1 receptors. For this, me