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流行病学研究提示,胃粘膜幽门螺杆菌 (Hp)感染与胃癌的发生有关。动物实验与体外实验发现 Hp慢性感染易引起胃粘膜细胞恶性转化 [1],分子生物学方面的研究亦证实 Hp感染与某些癌相关基因的改变存在相关关系 [2]。为从分子水平揭示 Hp感染在胃癌发生中的作用,本文采用 PCR技
Epidemiological studies suggest that gastric mucosal Helicobacter pylori (Hp) infection and the occurrence of gastric cancer. Animal experiments and in vitro experiments found that chronic infection of Hp easily lead to malignant transformation of gastric mucosal cells [1], molecular biology studies have confirmed that Hp infection and some cancer-related gene changes in the relationship [2]. In order to reveal the role of Hp infection in gastric carcinogenesis at molecular level, we used PCR technique