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本文系统介绍关于心肌梗塞时植物神经系统作用方面的研究进展。心肌梗塞时交感-儿茶酚胺活性增高,副交感神经兴奋性也同时提高。前者可使梗塞范围扩大,心室功能下降加剧及心律失常发生率增加,而后者对心肌梗塞具有保护作用。文中详细叙述上述作用的病理生理机制,并说明临床使用β阻滞剂及阿托品的原则及其原理。此外,心室壁感受器反射是心肌梗塞发生时机体的一种保护性机制。
This article systematically introduces the research progress on the role of autonomic nervous system in myocardial infarction. Sympathetic myocardial infarction - increased catecholamine activity, parasympathetic excitability also increased. The former can expand the scope of infarction, decreased ventricular dysfunction and increased incidence of arrhythmia, while the latter has a protective effect on myocardial infarction. The article describes in detail the pathophysiology of the above effects, and describes the clinical use of beta blockers and atropine principles and principles. In addition, ventricular wall reflex is a protective mechanism of the body when myocardial infarction occurs.