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目前认为,非甾体类抗炎药(ｎｏｎｓｔｅｒｏｉｄａｌａｎｔｉｉｎｆｌａｍｍａｔｏｒｙｄｒｕｇｓ,ＮＳＡＩＤｓ)主要通过抑制环氧化酶1(ｃｙｃｌｏｏｘｙｇｅｎａｓｅ1,ＣＯＸ1)活性,抑制了具有细胞保护作用的胃粘膜前列腺素的合成,诱发粘膜病变或促进消化性溃疡的发生[1]。局
Currently, nonsteroidal antiinflammatory drugs (NSAIDs) inhibit the synthesis of gastric mucosal prostaglandins, induce mucosal lesions or promote digestion by inhibiting the activity of cyclooxygenase 1 (COX1) The occurrence of ulcers [1]. Bureau