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患者女性,50岁,因反复心悸、胸闷半年入院,临床诊断:病毒性心肌炎。附图系1986年1月23日描记的心电图Ⅱ导联,示窦性心律、室性早搏呈二联律、室性早搏的联律间距呈390毫秒与470毫秒短-长交替出现,而来见在390—470毫秒之间者;室性早搏之QRS波形态相同。其机理是由于折返径路内存在纵向分离而形成双径路,快径路的绝对不应期长于窦性心动周期(S_1-S_2),其上方共同径路存在着单向传导阻滞,亦即窦性冲动不能通过上方共同径路进入双径路、而来自双径路的冲动则可通过上方共同径路;慢径路之绝对不应期短于窦性心动周期,当
Female patient, 50 years old, due to repeated palpitations, chest tightness, admitted for six months, clinical diagnosis: viral myocarditis. The figure is January 23, 1986 trace of ECG Ⅱ lead, showed sinus rhythm, ventricular premature beriberi law, premature ventricular contractions 390 milliseconds and 470 milliseconds short - long alternately, and come See 390-470 milliseconds between; QRS waveforms of ventricular premature beats the same. The mechanism is due to the existence of reentry pathways separated longitudinally and the formation of dual pathways, the absolute path of fast path is longer than the sinus cardiogram cycle (S_1-S_2), the common path above the existence of one-way block, which sinus impulse Can not enter the dual path through the common path above, and the impulse from the dual path can go through the common path above; the absolute refractory period of the slow path is shorter than the sinus cardiac cycle, when