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作者在前期研究工作的基础上,进一步探讨喉鳞癌抗原诱导的TAK细胞的杀瘤机理。结果表明:TAK细胞除直接杀伤靶细胞外,还通过分泌肿瘤坏死因子间接杀伤靶细胞。电镜观察表明,靶细胞的死亡形式有两种:溶解坏死和凋亡。
On the basis of previous studies, the authors further explore the mechanism of tumorigenicity of TAK cells induced by laryngeal squamous cell carcinoma antigen. The results showed that TAK cells could directly kill target cells by secreting tumor necrosis factor in addition to directly killing the target cells. Electron microscopy showed that the target cell death in two forms: lytic necrosis and apoptosis.