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目的 :探讨颈动脉狭窄在缺血性脑卒中所起的作用。方法 :依据临床和 CT诊断标准 ,将 4 8例住院病人分为腔隙性脑梗死组 ( 2 7例 )和非腔隙性脑梗死组 ( 2 1例 )。两组病人均不含椎基底动脉系统和心源性栓塞所致的颈内动脉系统脑梗死及行两侧颈部血管彩色多谱勒超声显像仪检测。结果 :两组间脉压差和甘油三酯水平有显著差异 (分别 P<0 .0 0 1 ,P<0 .0 5) ;腔隙性脑梗死组病灶同侧的颈动脉狭窄 (≥ 50 % )发生率为 1 8.5% ( 5/2 7) ,非腔隙性脑梗死组则达 52 .3% ( 1 1 /2 1 ) ,两组间有显著差异 ( P<0 .0 5)。结论 :非腔隙性脑梗死者病灶同侧颈动脉的严重狭窄较腔隙性脑梗死更为多见 ;高血压和甘油三酯在伴有颈动脉严重狭窄两组病例中的致病机制可能不同。
Objective: To investigate the role of carotid artery stenosis in ischemic stroke. Methods: Forty-eight inpatients were divided into lacunar infarction group (27 cases) and non-lacunar infarction group (21 cases) according to clinical and CT diagnostic criteria. Both groups of patients were free of vertebrobasilar system and cardioembolism-induced cerebral infarction of the internal carotid artery and bilateral cervical vessels by color Doppler ultrasound imaging. Results: There were significant differences in pulse pressure and triglyceride levels between the two groups (P <0.01, P <0.05, respectively); ipsilateral carotid stenosis (≥ 50 in the lacunar infarct group %) Were 8.5% (5/2 7) in the non-lacunar infarction group, and 52.3% (11/2/1) in the non-lacunar infarction group, with significant difference between the two groups (P0.05) . Conclusion: Severe stenosis of ipsilateral carotid artery in patients with non-lacunar infarction is more common than that of lacunar infarction. The pathogenesis of hypertension and triglyceride may be associated with severe carotid stenosis different.