原发性醛固酮增多症(primary aldosteronism,PA)是最常见的继发性高血压,占高血压患者的5%~15%,以醛固酮相对自主高分泌,无法被高盐所抑制为特征[1-2]。与血压分级及病程相似的原发性高血压患者相比,醛固酮过量自主分泌患者心血管病的发病率及死亡率明显增加[3],同时更易合并糖脂代谢异常[4-5],因此PA的系统性诊断及特异性治疗成为临床 Primary aldosteronism (PA) is the most common secondary hypertension, accounting for 5% -15% of patients with hypertension. It is characterized by its relatively high autocrine secretion of aldosterone [1 -2]. Compared with patients with essential hypertension whose blood pressure classification and course of disease are similar, the incidence of cardiovascular disease and mortality in patients with over-autonomic aldosterone secretion are significantly increased [3], and more likely to be associated with abnormalities in glucose and lipid metabolism [4-5] The systematic diagnosis and specific treatment of PA become clinical