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10条狗分成神经植入组(Ⅰ组,n=6)和对照组(N组,n=4),切断左侧喉返神经、喉上神经造成单侧喉麻痹模型。Ⅰ组以左侧舌下神经袢主支的分支分别植入同测甲杓肌、环杓侧肌;N组不作神经修复术。5个月后,直接喉镜检查Ⅰ组声带均无明显萎缩;发音时见4条狗术侧声带内收明显,位于正中位,声门闭合无裂隙;2条狗左侧声带固定于声门裂隙位,内收不明显。深吸气时左声带均未见内收现象。N组左侧声带均萎缩,固定于旁正中位。Ⅰ组发音恢复正常,N组声嘶音调低。自发肌电图检查示
Ten dogs were divided into nerve implantation group (group Ⅰ, n = 6) and control group (n = 4, n = 4). The left recurrent laryngeal nerve and the laryngeal nerve were caused by unilateral laryngeal paralysis. In group Ⅰ, branches of the main branch of the left hypoglossal nerve were respectively implanted into the same testis and circumferior; and group N was not subjected to nerve repair. After 5 months, there was no obvious atrophy of vocal cord in direct laryngoscopy group Ⅰ. Four vocal folds were observed on the side of the vocal folds in the middle of the vocal fold, with no crack in the glottis. The vocal folds of the two dogs were fixed on the glottis Fissure position, adduction is not obvious. Deep inspiratory left vocal fold were no adduction. N group left vocal cords were shrinking, fixed in the next median. Group Ⅰ pronunciation returned to normal, N group sounder tone down. Spontaneous EMG examination shows