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作者统计1986年该科就诊患者10720例,其中除外噪声性耳聋、突聋及美尼尔氏病等已知原因所致者,计得感音性耳聋812例。812例中听力急剧增恶者59例,小儿居多占45例。研究这些病例的听力曲线、平均听阈及听力增恶频带,均未见统计学差异。导致小儿感音性耳聋进一步恶化的因素有:感染13例,头颅创伤6例;生活环境改变4例;助听器16例;原因不明15例。应用皮质激素治疗41例,有效者25例,但停药后再度恶化者10例。当时患儿血中内源性皮质激素值均降至1.0μg/dl以下(正常小儿血中皮质醇为5.0～16.0平均7.8±3.2μg/dl),似属停药后反跳现象。作者报告2例典
The author statistics in 1986 the department of 10720 patients treated patients, except for noise-induced deafness, sudden deafness and Meniere’s disease and other known causes, accounting for 812 cases of sensorineural hearing loss. Among 812 cases, 59 cases were acute hearing loss and increased hearing loss, while most children accounted for 45 cases. Hearing curves, average hearing threshold and hearing enhancement frequency bands in these cases were not statistically different. Lead to further deterioration of sensorineural hearing loss in children: infection in 13 cases, 6 cases of head trauma; living environment change in 4 cases; hearing aid in 16 cases; unexplained 15 cases. Application of corticosteroids in 41 cases, 25 cases were effective, but again after the withdrawal of worsening in 10 cases. At that time, the blood levels of endogenous cortical hormones in children were reduced to 1.0μg / dl or less (normal children with cortisol 5.0 ~ 16.0 average 7.8 ± 3.2μg / dl), seems to be a rebound phenomenon after withdrawal. The author reports 2 cases Code