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无晶体囊样黄斑水肿系一综合征,其临床特征为轻度眼内炎症,荧光血管造影下,可见视乳头及黄斑部毛细血管荧光渗漏。病理学所见为黄斑部毛细血管周围有圆形细胞浸润。应用荧光血管造影术还证实其发生率在白内障摘出术后4～6周高达60%。而在术后第1周则少于5%。大多数无晶体囊样黄斑水肿病例,在病变自发消退之后,可恢复正常视力,少数有或无黄斑结构破坏的慢性囊样黄斑水肿,其视力可有明显障碍。在无晶体囊样黄斑水肿的病因中,前列腺素的分泌可能起着一定的作用,消炎痛为前列腺素合成的有力抑制剂,临床用以治疗关节炎,而用该药治疗无晶体囊样黄斑水肿的疗效意见不一
No crystal cystoid macular edema is a syndrome, the clinical features of mild intraocular inflammation, fluorescein angiography, visual papilla and macular capillary fluorescence leakage. Pathology of macular capillaries seen around the circular cell infiltration. The use of fluorescent angiography also confirmed the incidence of cataract surgery in 4 to 6 weeks after surgery up to 60%. While less than 5% in the first week after surgery. Most cases of aphakic cystoid macular edema, spontaneous regression of the disease, can restore normal vision, a few with or without macular destruction of chronic cystic macular edema, the visual acuity may have obvious obstacles. In the etiology of aphakic cystoid macular edema, prostaglandin secretion may play a role, indomethacin is a potent inhibitor of prostaglandin synthesis, clinical treatment of arthritis, and the use of the drug in the treatment of aphakic cystoid macula Efficacy of edema disagree