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调查玉米GY220×1145组合的RIL群体109个家系(F10;11)及其亲本在2个环境下粗缩病抗性的表型值,运用RIL群体的主基因多基因模型进行遗传分析,探讨玉米粗缩病抗性遗传规律。结果表明:①2008年GY220/1145组合的RILs粗缩病抗性性状的最佳遗传模型为E-1-5模型,即2对加性-显性-上位性主基因+加性-显性-上位性多基因混合遗传;2009年最佳遗传模型为G-0模型,即3对加性-上位性主基因+加性-上位性多基因模型混合遗传。②各主基因效应值不同。③上位性总效应小于主基因总效应。④有单个上位性效应大于单个主基因效应的情形出现。⑤主基因遗传为主,多基因遗传为辅。
This study investigated the phenotypic value of the resistance to rough set disease in 109 families (F10; 11) of RIL population with maize (GY220 × 1145) and their parents in two environments. Genetic analysis was conducted using the multiple gene model of main gene of RIL population. Genetic Consequences of Resistance to Crude Disease. The results showed as follows: ① The best genetic model for RILs resistance of RILs in combination of GY220 / 1145 in 2008 was E-1-5 model, that is, 2 pairs of additive-dominance-epistatic major genes plus additive- Epistatic polygene mixed inheritance; the best genetic model in 2009 was G-0 model, that is, three pairs of additive-epistatic major plus additive-epistatic polygenes were inherited. The main gene effect of different values. ③ Epistatic total effect is less than the main effect of the main genes. ④ A single epistatic effect is greater than a single major gene effect occurs. ⑤ main gene heredity, multi-genetics supplemented.