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光动力学疗法是一种新的肿瘤冶疗手段,近年来发展迅速,受到广泛重视。但其肿瘤杀伤的体内作用机理尚未完全阐明。许多研究结果表明,光动力学治疗后肿瘤坏死主要是由于血管损伤引起的组织缺氧所致。光动力学作用后,早期常可见到明显的血管改变、血流减缓以至完全停滞。但目前对发生这种变化的原因所知甚少,本文选用小鼠肝癌(Hep A)与LA-795
Photodynamic therapy is a new method of cancer treatment. It has developed rapidly in recent years and has received extensive attention. However, the in vivo mechanism of its tumor killing has not yet been fully elucidated. Many studies have shown that tumor necrosis after photodynamic therapy is mainly due to tissue hypoxia caused by vascular injury. After photodynamic effects, obvious vascular changes, slowed blood flow, and complete stagnation are often seen in the early stage. However, little is known about the reasons for this change. In this article, mouse liver cancer (Hep A) and LA-795 were selected.