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痤疮的炎症机制虽经广泛研究,但仍不清楚。许多证据证明痤疮是痤疮杆菌起着致病作用,有人认为痤疮杆菌脂酶分解皮脂产生刺激性脂肪酸引起炎症。但是,支持这一观点的证据经不住严格的评论和近年来的实验证实。Puhvel等(1966)在严重的炎性痤疮患者体内发现有抗痤疮杆菌的补体结合性抗体。以前著者已曾发现在痤疮早期炎症抗体中有补体C_3沉积。本文拟进一步报告更为详细研究的结果。
Although the mechanism of inflammation of acne has been extensively studied, it remains unclear. There is a lot of evidence that acne plays a causative role as acne bacteria. Some people think that acne bacteria lipase breaks down sebum and produces irritating fatty acids that cause inflammation. However, the evidence supporting this view can not stand rigorous comments and experiments in recent years confirmed. Puhvel et al. (1966) found complement-resistant antibodies against P. acnes in patients with severe inflammatory acne. Previous authors have found that there is complement C_3 deposition in early inflammatory antibodies to acne. This article is intended to further report the results of a more detailed study.