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用白兔30只分三组作实验,以缺氧代谢产物作观察指标,从细胞代谢水平,对比观察肝脏在硝普钠控制性降压和出血性休克时,是否存在因组织缺血缺氧而造成缺氧代谢。实验发现,出血性休克组动物MAP降到49mmHg,持续60分钟后,动脉乳酸浓度显著增加,显示了机体组织严重缺血缺氧。动脉和肝静脉乳酸差代表了肝脏对乳酸差为2.6mg/dl,肝脏是摄取转化乳酸的场所,放血降
30 rabbits were divided into three groups for experiments, the hypoxia metabolites as an indicator, from the cell metabolism levels, compared to observe the liver in sodium nitroprusside-controlled hypotension and hemorrhagic shock, whether there is tissue ischemia and hypoxia Resulting in hypoxia metabolism. The experiment found that MAP in hemorrhagic shock group dropped to 49mmHg, lasted 60 minutes, the arterial lactate concentration increased significantly, showing a serious tissue hypoxia. Artery and hepatic venous lactic acid difference on behalf of the poor lactic acid liver 2.6mg / dl, the liver is the location of uptake and conversion of lactic acid, bleeding