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贝尔氏麻痹与Hunt氏综合征较难鉴别。继Aitken氏(1933)以后,已有许多学者报告,不仅Hunt氏综合征起因于带状疱疹感染,连贝尔氏麻痹也可发现此种感染,因而无论从症状学或病因学上都无法明确鉴别这两种疾患。本文就22例发病在14天以内的贝尔氏麻痹和14例Hunt氏综合征检查其听力和平衡功能,并用血清补体结合反应测定病毒血清抗体效价来探讨二者之间的差异。病毒感染情况:日本学者用血清补体结合反应测定病毒抗体效价,贝尔氏麻痹中约30～40％有病毒感染,其中约20％乃带状疱疹病毒感染,本文22例贝尔氏麻痹中,有10例即45.5％乃病毒感染,其中6例为与带状疱疹病毒有关的所谓单纯性带状疱疹,占贝尔氏麻痹的27.3％。
Bell’s paralysis and Hunt’s syndrome more difficult to identify. Following Aitken’s (1933), many scholars have reported that not only the symptomatology or etiology of Hunt’s syndrome not only causes the infection of herpes zoster but also of Bell’s palsy These two disorders. In this paper, 22 cases of onset of Bell’s palsy within 14 days and 14 cases of Hunt’s syndrome were examined hearing and balance function, and serum complement binding reaction to determine the serum antibody titer to explore the differences between the two. Viral infections: Japanese scholars using serum complement binding reaction to determine the titer of the virus antibody, Bell’s palsy in about 30 to 40% of the virus infection, of which about 20% of herpes zoster virus infection, 22 cases of Bell’s palsy in this article, there Ten cases, or 45.5%, were infected with the virus, of which six were called herpes simplex associated with the herpes zoster virus, accounting for 27.3% of Bell’s palsy.