各种肾功不全动物模型研究显示,尿蛋白排泄是部分取决于肾小球毛细血管的压力。临床的进一步研究已经证明,不改变肾小球毛细血管压力的心痛定,使尿蛋白排泄增加而导致肾功能不全。而降低肾小球毛细血管压力的硫氮(艹卓)年酮可减少尿蛋白的排泄,其作用类似于血管紧张素转换酶抑制剂对糖 Various animal models of renal insufficiency studies have shown that urinary protein excretion is partly due to glomerular capillary pressure. Further clinical studies have shown that do not change the glomerular capillary pressure of nifedipine, urinary protein excretion caused by increased renal insufficiency. While reducing the glomerular capillary pressure of sulfur nitrogen (艹 Zhuo) ketones can reduce urinary protein excretion, its role is similar to angiotensin-converting enzyme inhibitors of sugar