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胎儿窘迫后生育的婴儿其脐带血浆中的胃动素含量明显增高,推测后者可能刺激胎粪的排泄。进一步研究发现某些消化道激素在胎儿窘迫时可释放。提示这些激素在分娩期窒息的病理生理学中可能起作用。收集曾有胎儿窘迫的婴儿之脐静脉血浆,其中胎心有2种异常6例、3种异常2例(经证实胎心过缓或过速,Ⅰ型或Ⅱ型逸脱,搏动间隙减少)。5例婴儿在分娩前排泄胎粪,3例婴儿在出生时需复苏术。患婴与20例正常阴道分娩婴儿血浆标本均用放射免疫法检测:抑胃肽、胰高糖素、肠高糖素、胃动素、胃泌素、胰泌素、神经降压素(neurotensin)和胰多肽(pp)。
Fetal distress after giving birth to the baby’s umbilical cord plasma motilin levels were significantly higher, suggesting that the latter may stimulate excretion of meconium. Further study found that some gastrointestinal hormones can be released in fetal distress. Suggesting that these hormones may play a role in the pathophysiology of asphyxia during labor. Umbilical venous plasma was collected from infants who had fetal distress. There were 6 abnormalities of fetal heart rate in 2 cases and 2 abnormalities of 3 abnormalities (evidence of overdrive or overdrive of fetal heart rate, type 1 or type 2 escaping and reduction of pulsatile gap). Five infants were excreted with meconium prior to delivery and three infants were resuscitated at birth. Infants and 20 normal vaginal birth infants plasma samples were detected by radioimmunoassay: gastric inhibitory peptide, glucagon, ghrelin, motilin, gastrin, secretin, neurotensin (neurotensin ) And pancreatic polypeptide (pp).