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目的:旨在确定初发的低血糖在患严重胎儿期酸血症足月儿中的新生儿脑损伤发生中的潜在作用。方法: 对185名足月新生儿进行了回顾性列表调查分析,他们在1993年2月至2002年12月之间被收住入新生儿重症监护室时测脐动脉血pH<7.00。短期的神经系统结果的监测包括严重脑病的死亡及有或没有抽风的中度到重度脑病的证据。低血糖定义为初次血糖≤4 g/L。结果:40(22%)例婴儿发展成神经系统方面异常,包括14 例(34%)患有严重的缺血缺氧性脑病的婴儿,他们已经死亡,24例(59%)患有中等到重度缺血缺氧性脑病的婴儿及3例(7%)癫痫发作的婴儿。185例婴儿中有27 例(14.5%)初次血糖≤4 g/L,这其中的15例(56%)有
PURPOSE: To determine the potential role of hypoglycaemia in neonatal brain injury in term infants with severe fetal acidosis. METHODS: A retrospective list of 185 full-term neonates was surveyed and analyzed for umbilical arterial blood pH <7.00 between February 1993 and December 2002 when admitted to a neonatal intensive care unit. Short-term monitoring of neurological outcome includes evidence of severe encephalopathy deaths and moderate to severe encephalopathy with or without ventilation. Hypoglycemia is defined as initial blood glucose ≤ 4 g / L. RESULTS: Forty infants (40%) developed neurological abnormalities including 14 (34%) infants with severe hypoxic-ischemic encephalopathy who had died and 24 (59%) had moderate to severe Severe hypoxic-ischemic encephalopathy in infants and 3 (7%) infants with seizures. Of 185 infants, 27 (14.5%) had initial blood glucose ≤ 4 g / L, of which 15 (56%) had